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Oxidative Stress Mess

February 11, 2014

Fish with tumor caught in St. Lucie River near Melbourne, FL

As I have stared at that Indian River Lagoon for six months and at the US in general for a year along with weather patterns, I tried to develop an image in my mind of what the atmosphere could be doing to the waterways.  I already had a model that our atmosphere/quantum vacuum field was normally weakly ionizing, it gets us all after 80 years or so, give or take.  And during storms it appears much more ionizing (just look at the atmosphere during a storm).  So I at least could use my chemical engineering background to understand that the atmosphere around those radar towers must be penetrating the waterways and chronically ionizing dissolved oxygen from the waterways, making it very hard for those fish to breathe.  And once you have free radicals of oxygen running around, look out, they will gradually oxidize the surroundings through redox reactions, creating what we know as “rust” on metals.  This includes the “rusting” or “aging” and accelerated oxidation of all surrounding biology.

So let’s imagine that Indian River Lagoon is like our bloodstream, carrying dissolved oxygen to those fish just as it carries dissolved oxygen throughout our bodies and brain.  But let’s suppose that something has penetrated and ionized that dissolved oxygen, such that it is now combining or oxidizing everything in its surroundings, such as nitrogen and phosphorous in the lagoon and possibly nitrogen in a childs’ bloodstream, producing nitrous oxide and such as those free radicals cause oxidative stress and hypoxic conditions within the bloodstream and on surrounding biology. This is what I perceive is happening when you get multiple radars overlapping and they increase this chronic increase in weakly ionizing discharge from the atmosphere on all biology below.  So here is a highly cited paper on oxidative stress as an indicator in autism spectrum disorders.

Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with autism

Nineteen of the 20 children participating in the study were diagnosed with “regressive” autism (apparently normal development
until regression into autism between ages 1.5 and 3 y).On the basis of their abnormal metabolic profiles, we hypothesize
that an increased vulnerability to oxidative stress (environmental, intracellular, or both) and impaired methylation capacity may
contribute to the development and clinical manifestation of regressive autism.

Hypoxia is also recognized as a potential trigger/indicator for autism:

Autism spectrum disorders and fetal hypoxia in a population-based cohort: Accounting for missing exposures via Estimation-Maximization algorithm

Autism spectrum disorders (ASD) are associated with complications of pregnancy that implicate fetal hypoxia (FH); the excess of ASD in male gender is poorly understood. We tested the hypothesis that risk of ASD is related to fetal hypoxia and investigated whether this effect is greater among males.

Proposed Toxic and Hypoxic Impairment of a Brainstem Locus in Autism

Abstract: Electrophysiological findings implicate site-specific impairment of the nucleus tractus solitarius (NTS) in autism. This invites hypothetical consideration of a large role for this small brainstem structure as the basis for seemingly disjointed behavioral and somatic features of autism. The NTS is the brain’s point of entry for visceral afference, its relay for vagal reflexes, and its integration center for autonomic control of circulatory, immunological, gastrointestinal, and laryngeal function. The NTS facilitates normal cerebrovascular perfusion, and is the seminal point for an ascending noradrenergic system that modulates many complex behaviors. Microvascular configuration predisposes the NTS to focal hypoxia. A subregion—the “pNTS”—permits exposure to all blood-borne neurotoxins, including those that do not readily transit the blood-brain barrier. Impairment of acetylcholinesterase (mercury and cadmium cations, nitrates/nitrites, organophosphates, monosodium glutamate), competition for hemoglobin (carbon monoxide, nitrates/nitrites), and higher blood viscosity (net systemic oxidative stress) are suggested to potentiate microcirculatory insufficiency of the NTS, and thus autism.

Oxygen and glucose deprivation in autism and role of calcium signalling

Neonatal hypoxia has been hypothesised to play an etiological role in development of autism. In animal models, hypoxia and hypoxic brain lesions are associated with some of autism-related neurobehavioral symptoms, including withdrawal in social and novel situations, diminished exploration in novel fields and repetitive behaviours [14985924164827128075818].

Based upon my research, I think the Indian River Lagoon is indicative of what is occurring within the bloodstream of all biology subject to chronic, weakly ionizing radiation from the surrounding atmosphere due to those overlapping, high powered, pulsed microwave radars. Children and the elderly are at greater immediate risk but I think all biology is suffering over time.

2-11-14 Melbourne 3 2-11-14 Melbourne 2 2-11-14 Melbourne

2-11-14 NOAA NEXRAD Melbourne 2-11-14 Cape Canaveral 5 2-11-14 PAB 4

From → Biology, Geophysics

  1. Heck Stewart. I’m actually beginning to understand some of what you write about. Whoo-hoo!
    Keep it up please.
    ~ Eric

  2. Thanks. By explaining it to others I am understanding it better myself.

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